De Camilli Lab
@pdc-lab.bsky.social
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Laboratory of Pietro De Camilli - Department of Neuroscience at Yale University
"Impaired hematopoiesis and embryonic lethality at midgestation of mice lacking both lipid transfer proteins VPS13A and VPS13C" out now in PLOS Biology
journals.plos.org/plosbiology/...
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Impaired hematopoiesis and embryonic lethality at midgestation of mice lacking both lipid transfer proteins VPS13A and VPS13C
VPS13A and VPS13C are bridge-like lipid transport proteins with distinct subcellular localization and function, and their absence is linked with chorea-acanthocytosis and Parkinson's disease, respecti...
https://journals.plos.org/plosbiology/article?id=10.1371/journal.pbio.3003393
about 1 month ago
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"The bridge-like lipid transport protein VPS13C/PARK23 mediates ER–lysosome contacts following lysosome damage" out now in Nature Cell Biology
rdcu.be/ehbaL
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The bridge-like lipid transport protein VPS13C/PARK23 mediates ER–lysosome contacts following lysosome damage
Nature Cell Biology - Wang et al. show the recruitment of the lipid channel protein VPS13C and formation of VPS13C-dependent contacts between endoplasmic reticulum and lysosomes after lysosomal...
https://rdcu.be/ehbaL
7 months ago
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reposted by
De Camilli Lab
The Science of Parkinson's
7 months ago
New research from
@pdc-lab.bsky.social
& collaborators finds that the bridge-like lipid transport protein VPS13C/PARK23 mediates ER–lysosome contacts following lysosome damage;
#Parkinsons-associated
LRRK2 is also recruited but at later stage
www.nature.com/articles/s41...
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